High cholesterol has long been causing bad rap Poor heart health – But a new study suggests that a low-carb ketogenic diet may not be associated with cardiovascular disease.
This study was conducted by Lundquist Long-term keto diet He developed elevated LDL cholesterol levels (called the “bad” type).
All participants, except for elevated cholesterol, were “metabolicly healthy” and followed a key diet for an average of five years, according to an April 7 press release.
The risk of heart disease is higher because of these unhealthy lifestyle habits
They are all eligible for LMHR (slimming large responders), which shows the adoption of people Carbohydrate-restricted diet and experience a significant increase in cholesterol.
High cholesterol has long become bad for causing poor heart health – but a new study suggests a low-carb ketogenic diet may not be associated with cardiovascular disease. (iStock)
Using advanced cardiac imaging, the researchers found that traditional cholesterol markers (APOB and LDL-C) were not associated with changes in plaque levels in the heart artery or baseline heart disease within one year.
Instead, existing plaque levels appear to be a better predictor of future plaque accumulation.
“Due to the elevated LDL, this population — which increases LDL due to the metabolic health of ketosis, and does not automatically increase heart risk,” said Bret Scher, MD, MD, medical director of Baszucki Group, told FOX News Digital.
“I’m a cardiac surgeon, and that’s what I cook at dinner
“So, we should probably move from LDL and APOB to vascular imaging using CAC or CTA to better predict risk prediction and inform how or whether to deal with someone’s heart risk factors.”
The results of the study were published in the American College of Cardiology: The Journal of the School of Progress.
Using advanced cardiac imaging, the researchers found that traditional cholesterol markers (APOB and LDL-C) were not associated with changes in plaque levels in the heart artery or baseline heart disease within one year. (iStock)
Previous studies also showed that people who are LMHR qualified have coronary plaques similar to those with comparable groups with normal LDL levels, “emphasizing that an increase in LDL induced by a ketogenic diet may not indicate a higher risk of coronary plaques,” the researchers said.
This is the first study to isolate very high LDL and APOB as risk factors for heart disease, noted Dr. Nick Norwitz, head of research and independent researcher at Oxford University.
“All other human studies include people with metabolic dysfunction or congenital people Genetic causes High LDL,” he told Fox News Digital.
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Nowitz said the results seem to be with what most clinicians would predict as well as the doctors taught in medical training.
“Although these data do not prove that traditional understandings are ‘erroneous’, they do in themselves indicate that traditional models have a large blind spot.”
One doctor said this is the first prospective trial in a unique population where traditional guidelines are often labeled as “high risk.” (iStock)
According to Norwitz Cholesterol levels When predicting plaque process.
“So CAC scores can be used to layer patients and help personalize care,” he told Fox News Digital.
Scher notes that “ketogenic therapy” can effectively treat certain metabolic-related diseases, but some people are afraid to continue on the keto diet due to cholesterol.
“This study provides support that they don’t necessarily need to stop eating or treating cholesterol – instead, they can work with their own cholesterol Medical Team For a more personalized and appropriate cardiac test,” he advises.
Ask important questions
Dr. Ken Berry, Family Doctor, Diabetes expert In Tennessee, instead of participating in the research, he shared his ideas about what he called “groundbreaking” research.
“The study found no association between the progression of coronary plaque using high-resolution CT angiography, LDL-C, APOB, and coronary plaque,” he told Fox News Digital.
“Instead, the strongest predictor of plaque progression is pre-existing plaque, not cholesterol levels – leading researchers to conclude that ‘plaque is plaque, Apob does not.’”
Existing plaque levels appear to be a better predictor of future plaque accumulation, although some experts warn of limitations. (iStock)
Berry said this is the first prospective trial in a unique population where traditional guidelines are often labeled as “high risk,” raising important questions about how to assess cardiovascular risk in low carbs, High-fat diet.
“The obvious implication is that if very high APOB levels are not a good predictor of heart attack risk in this particular population, is this a good predictor in any group of people?” he said.
“As I doubt it is just the latest popular lab tests to avoid scaring people to eat the proper ones Human diet Rich in saturated fat? ”
Potential research limitations
Dr. Bradley Serwer, a cardiologist and chief medical officer at Vitalsolution, a Cincinnati-based company, conducted a review of the study and pointed to some potential limitations.
“The study has a limited scope and involves a low-risk population with short-term durations, which makes it a challenge to generalize the findings as a broader, more vulnerable population,” Serwer told Fox News Digital.
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“Although the goal of the study is to propose hypotheses about the effects of dietary cholesterol, it does not provide clear evidence or object to its significance.”
However, cardiologists do agree with the authors’ conclusion that “improving risk stratification tools” are crucial to identify individuals at higher risk for coronary artery disease.
The effect of a ketogenic diet can be measured using a ketosis meter. “I hope doctors will be able to take this study and treat this particular population differently from other patients, understanding the unique physiological state of ketosis and the metabolic benefits it provides,” one researcher said. (iStock)
“Our primary responsibility as physicians is to evaluate each patient individually and to develop the most appropriate long-term care plan through common decisions,” he added.
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Michelle Routstein, a registered dietitian in New York City who specializes in heart disease, notes that plaque formation is a multi-step process that can take years to proceed.
“The environment of the arteries needs to be conducive to plaque formation,” Lawrencetin, who is not part of the study, told Fox News digital numbers.
“This is a group of humans with extraordinary physiology.”
“For example, there is hypertensionThis is a subgroup excluded from the study and is more likely to cause APOB to be deposited more easily on the arterial walls. ”
“If someone already has a plaque in the artery and maintains an increase in LDL and APOB, it can develop into more plaques, as shown in this study.”
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“But, if someone is Metabolic healthat baseline, there are no plaques, only APOB and LDL levels are elevated, so the environment may not necessarily lead to plaque formation over a year. ”
Routhenstein noted that previous research has shown that high LDL and APOB high in a person’s life, often combined with inflammation, insulin resistance and/or oxidative stress, can increase the risk of plaque development.
“It is important to note that many people who are implementing a ketogenic diet and ignore high LDL and APOB levels are often unaware that they have soft plaque brewing,” said nutritionist. (iStock)
She added: “It is important to note that many people who are implementing a ketogenic diet and ignore high LDL and APOB levels are often unaware that they have soft plaque brewing.”
“So it can be harmful to suggest that they ignore LDL and APOB levels, especially in a world where heart disease is so common and remains the leading cause of death worldwide.”
Looking to the future
Sher said he hopes more researchers can be inspired In addition, this study and apply it to different populations.
“But for now, I hope that doctors will be able to take this study and treat this particular population differently from other patients, understand the unique physiological state of ketosis and the metabolic benefits it provides,” he said.
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Nowitz said that in addition to more studies evaluating risks in this population, the team hopes to further investigate the mechanisms of the lean mass high reactor (LMHR) phenotype.
“This is a group of humans with extraordinary physiology,” he added.
